By Allard C. van der Wal, Anton E. Becker (auth.), Ernst E. Van Der Wall, Paul K. Blanksma, Menco G. Niemeyer, Willem Vaalburg, Harry J. G. M. Crijns (eds.)
In Advanced Imaging in Coronary Artery Disease, the position of a number of imaging options in diagnosing atherosclerosis, evaluation of myocardial ischemia, myocardial viability, and center failure are largely mentioned. the problems derived from cardiac puppy are awarded in terms of the traditional thoughts, corresponding to echocardiography, SPECT and MRI. moreover, more moderen imaging concepts reminiscent of intracoronary ultrasound, electron beam computed tomography, and Raman spectroscopy are given extensive realization. the consequences of drug remedy, corresponding to anti-ischemic and lipid-lowering medicines, also are evaluated.
This e-book will support medical cardiologists, nuclear medication physicians, fellows in cardiology and nuclear medication, radiochemists, uncomplicated examine fellows, and technicians, in knowing the recent advances in medical cardiac PET.
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Extra resources for Advanced Imaging In Coronary Artery Disease: PET, SPECT, MRI, IVUS, EBCT
Angioscopic evaluation of incompletely obstructing coronary intraluminal filling defects: comparison to angiography. Cathet Cardiovasc Diagn 1994;33:323-9. Chapter 3 RAMAN SPECTROSCOPY OF ATHEROSCLEROSIS: TOWARDS REAL-TIME IN VIVO HISTOCHEMISTRY AND PATHOLOGY Tjeerd J. Romer, James F. J. Buschman Summary The progression and regression of atherosclerotic plaques appear to be related to the amount and type of lipids that accumulate in the intima of arteries. 1. 3 Although several therapies are available to treat atherosclerosis, diagnostic methods that reliably predict lesion progression do not exist.
Proc Nat Acad Sci USA 1995;92:402-6. Kovanen PT, Kaartinen M, Paavonen T. Infiltrates of activated mast cells at the site of coronary atheromatous erosion or rupture in myocardial infarction. Circulation1995;92:1084-88. Moreno PR, Falk E, Palacios IF, Newell JB, Fuster V, Fallon JT. Macrophage infiltration in acute coronary syndromes. Implications for plaque rupture. Circulation, 1994;90:775-8. Brown DL, Hibbs MS, Keaney M, Loushin C, Isner JM. Identification of 92 kD gelatinase in human coronary atherosclerotic lesions: Association of active enzyme synthesis with unstable angina.
Lancet 1989;2:941-4. Jonasson L, Holm J, Skalli 0, Bondjers G, Hansson GK. Regional accumulations of T-cells, macrophages, and smooth muscle cells in the human atherosclerotic plaque. Arteriosclerosis 1986;6: 131-8. Van der Wal AC, Das PK, Bentz van de Berg 0, Van der Loos CM, Becker AE. Atherosclerotic lesions in humans. In situ immunophenotypic analysis suggesting an immune mediated response. Lab Invest 1989;61: 166-70. Kaartinen M, Pentilla A, Kovanen PT. Accumulation of activated mast cells in the shoulder region of human coronary atheroma, the predilection site of atheromatous rupture.
Advanced Imaging In Coronary Artery Disease: PET, SPECT, MRI, IVUS, EBCT by Allard C. van der Wal, Anton E. Becker (auth.), Ernst E. Van Der Wall, Paul K. Blanksma, Menco G. Niemeyer, Willem Vaalburg, Harry J. G. M. Crijns (eds.)